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Cavitation Infections and the Systemic Damage that they can cause.

Updated: Mar 2

Alicia Stewart, Research Scientist and imaet User
Alicia Stewart, Research Scientist and imaet User

The purpose of this article is to track possible pathways of cavitation infections to understand its potential implication for poor oral health, but also systemic dysfunction.  These infections are notoriously difficult to detect and treat for a variety of reasons. In most cases, there are not any symptoms that would alert someone to it. Some people do experience tooth and jaw pain, swelling and heat near the infection site, but generally it goes undetected for decades. Traditional dentists do not perform the type of scan that detects cavitations, and the specialized dentists who do don’t take traditional insurance, further compounding the problems with detection and treatment of these infections. This is why the IMAET is a critically helpful tool in assessing cavitation markers.

 

The process for identifying cavitation infections usually begins in one of two ways. The first way is when the patient comes in with clear cavitation symptoms and a history of root canals and wisdom teeth removal.  The second way, which is vastly more common, certain symptoms are presenting that have not responded to standard treatments. Cavitations can be the root cause of many conditions including the following: anxiety/depression, TMJ, tinnitus, insomnia, emotional instability, tonsilitis, sympathetic dominance, premature aging, acid reflux, non-alcoholic fatty liver disease, interstitial cystitis, and irritable bowel. We can track and follow all the pathways in which cavitations could be contributing to these conditions.

 

Cavitation Location Markers. There are a variety of physiological markers that will be inhibited or overactive due to a cavitation infection.

 

  • The jawbone.  Specifically, the maxilla and mandible will house most of the infection. The inflammation will likely cause damage to the surrounding tendons, ligaments, muscles, and fascial networks.


  • Brain fissures. Cavitation infection can be present in many tissues, glandular and brain, including the collateral sulcus, the hippocampal sulcus, the temporal sulcus, and the lateral sulcus. The system of these fissures spans the entire anatomy of the brain, and their purpose is to expand the surface area of the brain diversifying its structure and function.


  • The trigeminal nerve intersects in the jaw where an infection would be, and directly connects to other areas like the salivary glands, the sinuses, and the parotid gland and to the eyes via the ophthalmic nerve. The trigeminal nerve root is also directly adjacent the mid brain structures. Since cavitations can include strep and staph, eye infections and styes can be a result.


  • There are several arteries that travel right through the regions that would contain a cavitation. These include the carotid, the temporal, the cerebral and the basilar. The glands and structures with arterial supply that are upstream from the jaw could directly be impacted by infection. If the pineal gland is affected, which is one of the glands directly upstream, insomnia, anxiety, and emotional instability can be the result. If melatonin production is inhibited, the hypothalamus will be directly impacted. Not only will sleep wake cycles be disrupted but neuronal health for the entire brain could be inhibited because melatonin is highly neuroprotective. The pituitary would also be affected by a cavitation. This would not only inhibit growth hormone, ACTH, TSH, and antidiuretic hormone, but also oxytocin. Oxytocin is essential for mitochondrial ATP production. It is not difficult to track the downstream effects of infection downregulating the pituitary. Thyroid disease, adrenal dysfunction, hormone imbalance and poor mitochondrial function are just to name a few.


  • Salivary glands. There are three salivary glands that are directly adjacent where a cavitation presents, the salivary gland, the submandibular gland, the parotid gland. Besides producing saliva, these glands produce certain digestive enzymes. 60% of the body’s amylase is produced by the salivary glands, which is the enzyme responsible for metabolizing starch. Infection of these glands causes low salivary amylase and puts extra burden on the pancreas. The salivary glands also produce lingual lipase, which metabolizes fat. This enzyme is responsible for 10-30% of the body’s fat metabolism. Poor saliva production causes a host of problems including dry mouth, tooth decay, throat irritation.


  • Parotid gland. The parotid gland is situated near the back of the jaw is situated near the facial nerve and auditory cortex. An infection traveling to this gland can cause multiple problems. A swollen parotid gland will directly impact the auditory nerve and the auditory canal. Tinnitus could be an indicator of a cavitation infection. The parotid gland is also connected to the muscles of the jaw. TMJ, clenching, grinding and other conditions such as oromandibular dystonia, could be the result of such an infection.

 

 Additional downstream conditions possibly caused by cavitations.

 

  • Pancreas related disorders. If the salivary glands are not producing enough amylase and lingual lipase, there is going to be extra burden placed on the pancreas, which should only be responsible for a portion of starch and fat metabolism. This can result in pancreatitis, food intolerance and even diabetic markers. It is also possible that inhibition of the pancreas in this way could lead to cancer. Pancreatic enzymes are responsible for regulating cell growth. A pancreas under extra metabolic burden might not be able to regulate cancer cells as well as it is designed to.


  • Non-Alcoholic Fatty Liver Disease. Since we have already tracked the possible effect of a cavitations on the pancreas and metabolic function, it is not a difficult leap to make to also correlate its implication in non-alcoholic fatty liver disease. It is the natural process for starch to be converted into fatty acid and get deposited onto the liver. These fatty deposits will then be mobilized and converted to VLDL , then HDL and shuffled all over the body to places like the brain and spinal cord and are essential for their structure and function. If the salivary glands are not doing their job of digesting 60% of ingested starch, there will result in higher levels of undigested starch entering the digestive tract. The pancreas will then be creating higher levels of triglycerides which then get deposited onto the liver as fatty deposits. The presence of extra fatty deposits in the liver. This can cause hepatic insulin resistance, and in turn downregulate lipoprotein lipase. So, the presence of extra fatty deposits in the liver causes poor conversion into the kind of cholesterol the brain and spinal cord need. Of course, insulin resistance alone can cause NAFLD, but the cavitation pathways could also cause this because of poor starch digestion.


  • Chronic sinusitis. One of the bacterial culprits in cavitation infections is staphylococci, which can become something called MARCONS, which is multiple antibiotic resistant coagulase negative staph. Bacteria are easily transported via the nerves and circulatory system to the sinuses. MARCONS are notoriously difficult to treat because they survive within biofilm. They also produce exotoxins which increases the harm done to the body.


  • Hip disfunction and pelvic floor disorders.  Staphylococci and MARCONS can also be directly related to hip joint disfunction. This is because the jaw and the hip are connected by fascia via the deep frontal line. Bacteria in and around the muscles of the jaw can be transported directly to the muscles of the hip. From here the bacteria can travel to the bone and connective tissue as well. Once the bacteria are in the pelvic floor, the uterus could easily be affected. Also, one of the least understood medical conditions could be caused by cavitations as well. Interstitial cystitis. Especially if a woman has had a hysterectomy, it is highly likely that a bacterial infection of the pelvic floor muscles could trigger IC and a condition called sinking bladder. This is further corroborated by the fact that another bacterial strain in cavitation infections is Klebsiella. Klebsiella is also one of the major offenders in IC. It is more than plausible that the fascia could have transported that bacterium from the jaw directly to the pelvic floor.

Cavitations can be visualized with x-rays and MRIs. The unique approach with the bioenergetic communication tool, the IMAET, is the ability to identify bioenergetic signatures (frequencies) of the pathogens present with the BioScan function and then provide a feedback to the Immune System with a focus onto those specific pathogens. The Immune System can then go to work and start eliminating some of these 'bad boys', that have been flying under the radar for some time.

 

The continued research into the possible downstream conditions that cavitation infections can cause is crucial. It can be a major driving factor for many chronic conditions, especially when patients are not responding to traditional treatments. More research is needed to encourage changes around dental practices and cavitation detection methods as well.


About the Writer:

Alicia is a Research Scientist for CellCore Biosciences and began her career by graduating with a Bachelor’s Degree in Biochemistry from the University of Alaska in 2005. Her research involves finding answers to chronic conditions and she is certified in QEEG brainwave analysis. Alicia is graduating this spring with a naturopathic doctors diploma. She has already launched her own wellness business and is pioneering a system that utilizes a combination of her high-level research ability and IMAET sessions for her clients.

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